Brief Article
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World J Gastroenterol. Aug 28, 2011; 17(32): 3716-3723
Published online Aug 28, 2011. doi: 10.3748/wjg.v17.i32.3716
Potential risk factors for nonalcoholic steatohepatitis related to pancreatic secretions following pancreaticoduodenectomy
Sun Choon Song, Seong Ho Choi, Dong Wook Choi, Jin Seok Heo, Woo Seok Kim, Min Jung Kim
Sun Choon Song, Seong Ho Choi, Dong Wook Choi, Jin Seok Heo, Woo Seok Kim, Min Jung Kim, Department of Surgery, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul 135-710, South Korea
Author contributions: Choi SH designed the research study; Song SC, Choi DW, Heo JS, Kim WS, and Kim MJ conducted the research; Song SC provided analytical tools and analyzed the data; and Song SC prepared the manuscript.
Correspondence to: Seong Ho Choi, MD, PhD, Department of Surgery, Samsung Medical Center, Sungkyunkwan University School of Medicine, 50 Irwon-dong, Gangnam-gu, Seoul 135-710, South Korea. sh3468.choi@samsung.com
Telephone: +82-2-3410-3468 Fax: +82-2-3410-6980
Received: March 3, 2011
Revised: April 16, 2011
Accepted: April 23, 2011
Published online: August 28, 2011
Abstract

AIM: To identify risk factors for nonalcoholic steatohepatitis following pancreaticoduodenectomy, with a focus on factors related to pancreatic secretions.

METHODS: The medical records of 228 patients who had a pancreaticoduodenectomy over a 16-mo period were reviewed retrospectively. The 193 patients who did not have fatty liver disease preoperatively were included in the final analysis. Hepatic steatosis was diagnosed using the differences between splenic and hepatic attenuation and liver-to-spleen attenuation as measured by non-enhanced computed tomography.

RESULTS: Fifteen patients (7.8%) who showed postoperative hepatic fatty changes were assigned to Group A, and the remaining patients were assigned to Group B. Patient demographics, preoperative laboratory findings (including levels of C-peptide, glucagon, insulin and glucose tolerance test results), operation types, and final pathological findings did not differ significantly between the two groups; however, the frequency of pancreatic fistula (P = 0.020) and the method of pancreatic duct stenting (P = 0.005) showed significant differences between the groups. A multivariate analysis identified pancreatic fistula (HR = 3.332, P = 0.037) and external pancreatic duct stenting (HR = 4.530, P = 0.017) as independent risk factors for the development of postoperative steatohepatitis.

CONCLUSION: Pancreatic fistula and external pancreatic duct stenting were identified as independent risk factors for the development of steatohepatitis following pancreaticoduodenectomy.

Keywords: Nonalcoholic fatty liver diseases; Nonalco-holic steatohepatitis; Pancreatic duct stenting; Pancreatic fistula; Pancreatic surgery