Brief Article
Copyright ©2010 Baishideng Publishing Group Co., Limited. All rights reserved.
World J Gastroenterol. Dec 28, 2010; 16(48): 6128-6134
Published online Dec 28, 2010. doi: 10.3748/wjg.v16.i48.6128
Tissue factor in predicted severe acute pancreatitis
Ellen Andersson, Jakob Axelsson, Gunilla Eckerwall, Daniel Ansari, Roland Andersson
Ellen Andersson, Jakob Axelsson, Gunilla Eckerwall, Daniel Ansari, Roland Andersson, Department of Surgery, Clinical Sciences Lund, Lund University and Lund University Hospital, SE-221 85 Lund, Sweden
Author contributions: Andersson E designed the experiment, calculated data, wrote the paper; Axelsson J set up and performed the ELISA analyses; Eckerwall G included patients and performed ELISA analyses; Ansari D contributed in writing the paper and analysing data; Andersson R took part in the design of the experiment.
Supported by The Skane County Council Research and Development Foundation, No. REGSKANE-61401; and the Erik and Angelica Sparre Foundation, No. 081230
Correspondence to: Ellen Andersson, MD, Department of Surgery, Clinical Sciences Lund, Lund University and Lund University Hospital, SE-221 85 Lund, Sweden. ellen.andersson@med.lu.se
Telephone: +44-121-4142972 Fax: +44-121-6272384
Received: June 18, 2010
Revised: August 30, 2010
Accepted: September 7, 2010
Published online: December 28, 2010
Abstract

AIM: To study tissue factor (TF) in acute pancreatitis and evaluate the role of TF as a predictive marker of severity.

METHODS: Forty-nine consecutive patients admitted to Lund University Hospital, fulfilling the criteria of predicted severe acute pancreatitis (AP), were recruited prospectively between 2002 and 2004. Blood samples for TF analyses were drawn at inclusion in the study and 12 h, 1 d and 3 d later.

RESULTS: Twenty-seven patients developed mild AP, and 22 patients severe AP. At inclusion in the study, the groups were comparable with respect to gender, aetiology, Acute Physiology and Chronic Health Evaluation II score, and duration of pain. At inclusion in the study and at 12 h, TF was higher in the severe AP group (P = 0.035 and P = 0.049, respectively). After 1 and 3 d, no differences in TF levels were noted. Interleukin (IL)-6 was significantly higher in the severe AP group at all of the studied time points. C-reactive protein (CRP) was significantly higher in the AP group at 1 and 3 d. In receiver operating characteristic-curves, the area under the curve (AUC) for TF was 0.679 (P = 0.035) at inclusion in the study, and a cut off level for TF of 40 pg/mL showed a sensitivity of 71% and a specificity of 67%, whereas corresponding AUC for IL-6 was 0.775, P = 0.001, and for CRP was 0.653. IL-6 showed better AUC-values than TF at all time points studied.

CONCLUSION: TF-levels are raised early in severe AP. TF as an early predictive marker of severe AP is superior to CRP, but inferior to IL-6.

Keywords: Acute pancreatitis; Coagulation; Prediction of severity; Tissue factor