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World J Gastroenterol. Sep 28, 2008; 14(36): 5606-5611
Published online Sep 28, 2008. doi: 10.3748/wjg.14.5606
Tumor suppress genes screening analysis on 4q in sporadic colorectal carcinoma
Li-Xin Jiang, Jie Xu, Zhao-Wen Wang, Da-Peng Li, Zhi-Hai Peng, Jian-Jun Gao, Lin He, Hai-Tao Zheng
Li-Xin Jiang, Jie Xu, Hai-Tao Zheng, Department of Abdomen Surgery Department, the Affiliated Yantai Yuhuangding Hospital of Qingdao University Medical College, Yantai 264000, Shandong Province, China
Zhao-Wen Wang, Da-Peng Li, Zhi-Hai Peng, Department of General Surgery, the Affiliated Shanghai First People Hospital of Shanghai Jiaotong University, Shanghai 200080, China
Jian-Jun Gao, Lin He, Shanghai Bio-x Center, Shanghai Jiaotong University, Shanghai 200031, China
Author contributions: He L, Peng ZH designed research; Jiang LX, Xu J, Zheng HT performed research; Gao JJ contributed new reagents; Wang ZW and Li DP analyzed data; and Jiang LX, Zheng HT wrote the paper.
Supported by The National Natural Science Foundation of China, No. 30080016 and No. 30470977
Correspondence to: Hai-Tao Zheng, Department of Abdomen Surgery Department, the Affiliated Yantai Yuhuangding Hospital of Qingdao University Medical College, Yantai 264000, Shangdong Province, China. zhenghaitao1972@126.com
Telephone: +86-535-6691999-81707 Fax: +86-535-6691999
Received: July 20, 2008
Revised: August 26, 2008
Accepted: September 3, 2008
Published online: September 28, 2008
Abstract

AIM: To search candidate tumor suppressor genes (TSGs) on chromosome 4q through detecting high loss of heterozygosity (LOH) regions in sporadic colorectal carcinoma in Chinese patients.

METHODS: Thirteen fluorescent labeled polymorphic microsatellite markers were analyzed in 83 cases of colorectal carcinoma and matched normal tissue DNA by polymerase chain reaction (PCR). PCR products were electrophoresed on an ABI 377 DNA sequencer. Genescan 3.7 and Genotype 3.7 software were used for LOH scanning and analysis. Comparison between LOH frequency and clinicopathological factors were performed by χ2 test.

RESULTS: Data were collected on all informative loci. The average LOH frequency on 4q was 28.56%. The D4S2915 locus showed highest LOH frequency (36.17%). Two obvious deletion regions were detected: one between D4S3000 and D4S2915 locus (4q12-21.1), another flanked by D4S407 and D4S2939 locus (4q25-31.1). None case showed complete deletion of 4q, most cases displayed interstitial deletion pattern solely. Furthermore, compared with clinicopathological features, a significant relationship was observed between LOH frequencies on D4S3018 locus. In tumors larger than 5 cm in diameter, LOH frequency was significantly higher than tumors that were less than 5 cm (56% vs 13.79%, P = 0.01). On D4S1534 locus, LOH was significantly associated with liver metastasis (80% vs 17.25%, P = 0.012). No relationship was detected on other locus compared with clinicopathological features.

CONCLUSION: By high resolution deletion mapping, two high frequency regions of LOH (4q12-21.1 and 4q25-31.1) were detected, which may contribute to locate TSGs on chromosome 4q involved in carcinogenesis and progression of sporadic colorectal carcinoma.

Keywords: Loss of heterozygosity; Colorectal carcinoma; Chromosome; 4q; Tumor suppressor gene