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World J Gastroenterol. Nov 21, 2006; 12(43): 7019-7024
Published online Nov 21, 2006. doi: 10.3748/wjg.v12.i43.7019
Hepatitis B genotypes: Relation to clinical outcome in patients with chronic hepatitis B in Saudi Arabia
Ayman A Abdo, Badr M Al-Jarallah, Faisal M Sanai, Ahmad S Hersi, Khalid Al-Swat, Nahla A Azzam, Manal Al-Dukhayil, Amira Al-Maarik, Faleh Z Al-Faleh
Ayman A Abdo, Ahmad S Hersi, Khalid Al-Swat, Nahla A Azzam, Manal Al-Dukhayil, Amira Al-Maarik, Faleh Z Al-Faleh, Gastroenterology Division, Faculty of Medicine, King Saud University, Riyadh, Saudi Arabia
Faisal M Sanai, Hepatology Division, Department of Medicine, Riyadh Military Hospital, Riyadh, Saudi Arabia
Badr M Al-Jarallah, Gastroenterology Unit, Department of Medicine, McGill University, Montreal, Canada
Author contributions: All authors contributed equally to the work.
Supported by a grant from the Research Center at the College of Medicine, King Saud University, Riyadh
Correspondence to: Dr. Ayman A Abdo, College of Medicine, King Saud University, PO Box 2925 (59), Riyadh 11461, Saudi Arabia. abdoayman@hotmail.com
Telephone: +966-1-4671215 Fax: +966-1-4671217
Received: August 27, 2006
Revised: September 15, 2006
Accepted: September 27, 2006
Published online: November 21, 2006
Abstract

AIM: To identify the most common hepatitis B virus (HBV) genotype in Saudi Arabia, and correlate the prevailing genotypes with the clinical outcome of patients.

METHODS: Patients were consecutively recruited from the hepatology clinics of two tertiary care referral centers. Patients were categorized into 4 different groups: group 1, patients with hepatitis B and normal liver enzymes; group 2, patients with hepatitis B and abnormal liver enzymes but without cirrhosis; group 3, patients with hepatitis B and liver cirrhosis; group 4, patients with hepatitis B and hepatocellular carcinoma. All patients had a positive hepatitis B surface antigen (HBsAg). Genotyping of HBV was performed by nested PCR-mediated amplification of the target sequence and hybridization with sequence-specific oligonucleotides.

RESULTS: Seventy patients were enrolled in this study. They were predominantly male (72.9%) in their mid-forty’s (mean age 47 years). Forty-nine (70%) patients were hepatitis B envelope antigen (HBeAg) negative. The majority of patients (64%) acquired HBV through unknown risk factors. Hepatitis B genotyping revealed that 57 patients (81.4%) were genotype D, 1 patient (1.4%) had genotype A, 1 patient (1.4%) had genotype C, and 4 patients (5.7%) had genotype E, while 7 patients (10%) had mixed genotype (4 patients ADG, 1 patient DE, 1 patient DF, and 1 patient ADFG). Based on univariate analysis of genotype D patients, significant predictors of advanced liver disease were age, gender, aspartate transaminase, alanine transaminase, albumin, bilirubin, and alkaline phosphatase (all P < 0.001). In multivariate analysis decreased hemoglobin (r = -0.05; 95% CI: -0.08 to -0.03; P = 0.001) and albumin levels (r = -0.004; 95% CI: -0.007 to -0.001; P = 0.002) were highly significant predictors of advanced liver disease. In patients with HBV genotype D, HBeAg negativity was found to increase across advancing stages of liver disease (P = 0.024).

CONCLUSION: This study highlights that the vast majority of Saudi patients with chronic hepatitis B have genotype D. No correlation could be observed between the different genotypes and epidemiological or clinical factors. The relationship between genotype D and HBeAg status in terms of disease severity needs to be further elucidated in larger longitudinal studies.

Keywords: Hepatitis B virus; Genotype D; Cirrhosis; Hepatocellular carcinoma; Saudi Arabia