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World J Gastroenterol. May 28, 2006; 12(20): 3275-3282
Published online May 28, 2006. doi: 10.3748/wjg.v12.i20.3275
Direct ex vivo analysis of dendritic cells in patients with hepatocellular carcinoma
Lars A Ormandy, Anatol Färber, Tobias Cantz, Susanne Petrykowska, Heiner Wedemeyer, Monique Hörning, Frank Lehner, Michael P Manns, Firouzeh Korangy, Tim F Greten
Lars A Ormandy, Anatol Färber, Tobias Cantz, Susanne Petrykowska, Heiner Wedemeyer, Monique Hörning, Michael P Manns, Firouzeh Korangy, Tim F Greten, Department of Gastroenterology, Hepatology and Endocrinology, Hannover Medical School, Germany
Frank Lehner, Departments of Visceral and Transplantation Surgery, Hannover Medical School, Germany
Author contributions: All authors contributed equally to the work.
Supported by Erich und Gertrud Roggenbruck Stiftung, Deutsche Forschungsgemeinschaft (KFO 119)
Correspondence to: Dr. Tim F Greten, Department of Gastroenterology, Hepatology und Endocrinology, Medizinische Hochschule Hannover, Carl Neuberg Str. 1, 30625 Hannover, Germany. greten.tim@mh-hannover.de
Telephone: +49-511-5328941 Fax: +49-511-5328945
Received: December 12, 2005
Revised: January 8, 2006
Accepted: January 14, 2006
Published online: May 28, 2006
Abstract

AIM: To analyze the phenotype and function of dendritic cells (DC) from patients with hepatocellular carcinoma (HCC) in order to understand their role in this disease.

METHODS: Myeloid dendritic cells were enumerated in peripheral blood of HCC patients. CD80, CD83, CD86 and HLA-DR expression on naïve and stimulated myeloid dendritic cells from peripheral blood were analyzed. Myeloid dendritic cells were isolated from peripheral blood and their function was tested. Phagocytosis was analyzed using FITC-dextran beads, peptide specific stimulation, the capacity to stimulate allogeneic T cells and secretion of cytokines upon poly dI:dC was tested.

RESULTS: Myeloid dendritic cells were reduced in patients with HCC. No differences in CD80, CD83, CD86 and HLA-DR expression were found on naïve and stimulated myeloid dendritic cells from HCC patients and healthy controls. Normal phagocytosis or stimulation of peptide specific T cells was observed in contrast to an impaired allo-stimulatory capacity and a reduced IL-12 secretion.

CONCLUSION: Impaired IL-12 production of mDCs in patients could lead to an impaired stimulatory capacity of naïve T cells suggesting that IL-12 directed therapies may enhance tumor specific immune responses in HCC patients.

Keywords: Myeloid DC; Hepatocellular carcinoma; CD80; CD83; CD86