Rapid Communication
Copyright ©2005 Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Dec 28, 2005; 11(48): 7657-7660
Published online Dec 28, 2005. doi: 10.3748/wjg.v11.i48.7657
Asthma and gastroesophageal reflux disease: Effect of long-term pantoprazole therapy
Calabrese Carlo, Fabbri Anna, Areni Alessandra, Scialpi Carlo, Zahlane Desiree, Di Febo Giulio
Calabrese Carlo, Fabbri Anna, Areni Alessandra, Scialpi Carlo, Zahlane Desiree, Di Febo Giulio, Department of Internal Medicine and Gastroenterology, University of Bologna, Italy
Author contributions: All authors contributed equally to the work.
Supported by grants from Altana-Pharma Italia
Correspondence to: Carlo Calabrese, Department of Internal Medicine and Gastroenterology, Policlinico S. Orsola-Malpighi, Via Massarenti 9, 40138 Bologna, Italy. calabrese.c@med.unibo.it
Telephone: +390516364191 Fax: +390516364138
Received: March 1, 2005
Revised: August 1, 2005
Accepted: August 3, 2005
Published online: December 28, 2005
Abstract

AIM: To define the prevalence of gastroesophageal reflux disease (GERD) in mild persistent asthma and to value the effect of pantoprazole therapy on asthmatic symptoms.

METHODS: Seven of thirty-four asthmatic patients without GERD served as the non-GERD control group. Twenty-seven of thirty-four asthmatic patients had GERD (7/27 also had erosive esophagitis, sixteen of them presented GERD symptoms. An upper gastrointestinal endoscopy was performed in all the subjects to obtain five biopsy specimens from the lower 5 cm of the esophagus. Patients were considered to have GERD when they had a dilation of intercellular space (DIS)>0.74 μm at transmission electron microscopy. Patients with GERD were treated with pantoprazole, 80 mg/day. Forced expiratory volume in one second (FEV1) was performed at entry and after 6 mo of treatment. Asthmatic symptoms were recorded. The required frequency of inhaling rapid acting β2-agonists was self-recorded in the patients’ diaries.

RESULTS: Seven symptomatic patients presented erosive esophagitis. Among the 18 asymptomatic patients, 11 presented DIS, while all symptomatic patients showed ultrastructural esophageal damage. Seven asymptomatic patients did not present DIS. At entry the mean of FEV1 was 1.91 L in symptomatic GERD patients and 1.88 L in asymptomatic GERD patients. After the treatment, 25 patients had a complete recovery of DIS and reflux symptoms. Twenty-three patients presented a regression of asthmatic symptoms with normalization of FEV1. Four patients reported a significant improvement of symptoms and their FEV1 was over 80%.

CONCLUSION: GERD is a highly prevalent condition in asthma patients. Treatment with pantoprazole (80 mg/day) determines their improvement and complete regression.

Keywords: Asthma; Gastroesophageal reflux disease; Pantoprazole; NERD; ERD; Dilated intercellular spaces; TEM