Brief Reports
Copyright ©The Author(s) 2005. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jul 21, 2005; 11(27): 4268-4271
Published online Jul 21, 2005. doi: 10.3748/wjg.v11.i27.4268
Genetic polymorphisms of N-acetyltransferase 2 and colorectal cancer risk
Lu-Jun He, Yue-Ming Yu, Fang Qiao, Jing-Shan Liu, Xiao-Feng Sun, Ling-Ling Jiang
Lu-Jun He, Ling-Ling Jiang, Department of Biochemistry, Hebei Medical University, Shijiazhuang 050017, Hebei Province, China
Fang Qiao, Jing-Shan Liu, Laboratory of HLA, Hebei Province Blood Center, Shijiazhuang 050071, Hebei Province, China
Yue-Ming Yu, Department of Surgery, No. 4 Hospital of Hebei Medical University, Shijiazhuang 050011, Hebei Province, China
Xiao-Feng Sun, Department of Oncology, Institute of Biomedicine and Surgery, University of Linköping, S-581 85 Linköping, Sweden
Author contributions: All authors contributed equally to the work.
Correspondence to: Dr. Ling-Ling Jiang, Department of Biochemistry, Hebei Medical University, Shijiazhuang 050017, Hebei Province, China. guiyang1959@yahoo.com
Telephone: +86-311-6265639 Fax: +86-311-7061014
Received: October 19, 2004
Revised: November 23, 2004
Accepted: November 26, 2004
Published online: July 21, 2005
Abstract

AIM: To identify the distribution of N-acetyltrasferase 2 (NAT2) polymorphism in Hebei Han Chinese and the effects of the polymorphism on the development of colorectal cancer.

METHODS: We performed a hospital-based case-control study of 237 healthy individuals and 83 colorectal cancer patients of Hebei Han Chinese. DNA was extracted from peripheral blood and cancer tissues. The genotypes of the polymorphisms were assessed by PCR-restriction fragment length polymorphism(RFLP).

RESULTS: There were four NAT2 alleles of WT, M1, M2, and M3 both in the healthy subjects and in the patients, and 10 genotypes of WT/WT, WT/M1, WT/M2, WT/M3, M1/M1, M1/M2, M1/M3, M2/M2, M2/M3, M3/M3. M2 allele was present in 15.61% of healthy subjects and 29.52% of patients (χ2 = 15.31, P < 0.0001), and M3 allele was present in 30.59% of healthy subjects and 16.87% of patients (χ2 = 25.33, P < 0.0001). There were more WT/M2 (χ2 = 34.42, P < 0.0001, odd ratio = 4.99, 95%CI = 2.27-9.38) and less WT/M3 (χ2 = 3.80, P = 0.03) in the patients than in the healthy subjects. In 70.3% of the patients, there was a difference in NAT2 genotype between their tumors and blood cells. Patients had more WT/M2 (χ2 = 5.11, P = 0.02) and less M2/M3 (χ2 = 4.27, P = 0.039) in their blood cells than in the tumors. Furthermore, 53.8% (7/13) of M2/M3 in tumors were from WT/M2 of blood cells.

CONCLUSION: There is a possible relationship between the NAT2 polymorphisms and colorectal cancer in Hebei Han Chinese. The genotype WT/M2 may be a risk factor for colorectal cancer.

Keywords: NAT2 gene; Colorectal cancer; RFLP