Brief Reports
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Mar 1, 2004; 10(5): 750-754
Published online Mar 1, 2004. doi: 10.3748/wjg.v10.i5.750
Pathobiological behavior and molecular mechanism of signet ring cell carcinoma and mucinous adenocarcinoma of the stomach: A comparative study
Xue-Fei Yang, Lin Yang, Xiao-Yun Mao, Dong-Ying Wu, Su-Min Zhang, Yan Xin
Xue-Fei Yang, Xiao-Yun Mao, Dong-Ying Wu, Su-Min Zhang, Yan Xin, The Fourth Laboratory, Cancer Institute, The First Affiliated Hospital of China Medical University, Shenyang 110001, Liaoning Province, China
Lin Yang, Department of Pathology, Cancer Hospital, Chinese Academy of Medical Sciences, Beijing 100021, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No.30070845 and No.30371607
Correspondence to: Professor Yan Xin, The Fourth Laboratory, Cancer Institute, The First Affiliated Hospital of China Medical University, Shenyang 110001, Liaoning Province, China. yxin@mail.cmu.edu.cn
Telephone: +86-24-23256666 Ext 6351 Fax: +86-24-23253443
Received: September 9, 2003
Revised: October 15, 2003
Accepted: October 22, 2003
Published online: March 1, 2004
Abstract

AIM: To elucidate the distinctive pathobiological behavior between signet ring cell carcinoma (SRC) and mucinous adenocarcinoma of the stomach.

METHODS: Based on the histological growth patterns and cell-functional differentiation classifications of stomach carcinoma, we conducted a series of comparative studies. All paraffin-embedded and frozen blocks were collected from the files of Cancer Institute of China Medical University. On the basis of histopathological observation, we applied enzymatic and mucous histochemistry, immunohistochemistry, flow cytometry (FCM) and molecular biology to compare these two categories of gastric cancers in terms of the DNA ploidy, proliferative kinetics, the expression of gastric carcinoma associated gene product and instabilities of mitochondrial DNA (mtDNA).

RESULTS: Gastric SRC was commonly seen in females below 45 years, mostly presenting diffuse growth and ovary or uterine cervix metastasis. The majority of SRC were absorptive and mucus-producing functional differentiation type (AMPFDT), which growth relied on estrogen. Meanwhile, stomach mucinous adenocarcinomas were mostly observed in males over 50 years, prone to massive growth or nest growth and extensive peritoneal infiltration, showing two categories of cell-functional differentiation types: AMPFDT and mucus-secreting functional differentiation type (MSFDT). Expressions of ER, enzyme c-PDE and 67 kDa LN-R in SRC were evidently higher than that in mucinous adenocarcinoma, while expressions of LN, CN-IV, CD44v6, and PTEN protein were obviously lower in SRC than that in mucinous adenocarcinoma (P < 0.05). There was no statistic significance in VEGF, ECD and instabilities of mtDNA (P > 0.05) between the above two gastric carcinomas.

CONCLUSION: Though SRC and mucinous adenocarcinoma were both characterized by abundant mucus-secretion, they were quite different in morphology, ultrastructure, cell-functional differentiation and protein expression, indicating different mechanisms of carcinogenesis. We concluded that combining histological growth patterns, cell-functional differentiation types with tumor related markers might be significant in early diagnosis and prognosis assessment for SRC and mucinous adenocarcinoma of the stomach.

Keywords: $[Keywords]