H Pylori
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Nov 15, 2004; 10(22): 3274-3277
Published online Nov 15, 2004. doi: 10.3748/wjg.v10.i22.3274
Neither gastric topological distribution nor principle virulence genes of Helicobacter pylori contributes to clinical outcomes
Yan Wing Ho, Khek Yu Ho, Felipe Ascencio, Bow Ho
Yan Wing Ho, Bow Ho, Department of Microbiology, Faculty of Medicine, National University of Singapore, Singapore
Khek Yu Ho, Department of Medicine, Faculty of Medicine, National University of Singapore, Singapore
Felipe Ascencio, Departamento de Patologia Marina, Centro de Investigaciones Biologicas del Noroeste (CIBNOR), La Paz, Mexico
Yan Wing Ho, is a National University of Singapore Research Scholar, Singapore
Author contributions: All authors contributed equally to the work.
Supported by NMRC Grant , No. 0415/2000, R-182-000-037-213
Correspondence to: Bow Ho, Department of Microbiology, Faculty of Medicine, National University of Singapore, 5 Science Drive 2, Singapore 117597, Republic of Singapore. michob@nus.edu.sg
Telephone: +65-68743672 Fax: +65-67766872
Received: February 27, 2004
Revised: April 4, 2004
Accepted: April 29, 2004
Published online: November 15, 2004
Abstract

AIM: Studies on Helicobacter pylori (H pylori) and gastrodu- odenal diseases have focused mainly on the distal sites of the stomach, but relationship with the gastric cardia is lacking. The aim of this study is to determine if the gastric topology and genotypic distribution of H pylori were associated with different upper gastrointestinal pathologies in a multi-ethnic Asian population.

METHODS: Gastric biopsies from the cardia, body/corpus and antrum were endoscoped from a total of 155 patients with dyspepsia and/or reflux symptoms, with informed consent. H pylori isolates obtained were tested for the presence of 26kDa, ureC, cagA, vacA, iceA1, iceA2 and babA2 genes using PCR while DNA fingerprints were generated using random amplification polymorphic DNA (RAPD).

RESULTS: H pylori was present in 51/155 (33%) of patients studied. Of these, 16, 15 and 20 were isolated from patients with peptic ulcer diseases, gastroesophageal reflux diseases and non-ulcer dyspepsia, respectively. Of the H pylori positive patients, 75% (38/51) had H pylori in all three gastric sites. The prevalence of various genes in the H pylori isolates was shown to be similar irrespective of their colonization sites as well as among the same site of different patients. The RAPD profiles of H pylori isolates from different gastric sites were highly similar among intra-patients but varied greatly between different patients.

CONCLUSION: Topographic colonization of H pylori and the virulence genes harboured by these isolates have no direct bearing to the clinical state of the patients. In multi-ethnic Singapore, the stomach of each patient is colonized by a predominant strain of H pylori, irrespective of the clinical diagnosis.

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