Liver Cancer
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jul 15, 2004; 10(14): 2019-2023
Published online Jul 15, 2004. doi: 10.3748/wjg.v10.i14.2019
Effects of KAI1 gene on growth and invasion of human hepatocellular carcinoma MHCC97-H cells
Sui-Hai Si, Jian-Min Yang, Zhi-Hong Peng, Yuan-Hui Luo, Ping Zhou
Sui-Hai Si, Jian-Min Yang, Zhi-Hong Peng, Yuan-Hui Luo, Ping Zhou, Gastroenterology Research Center, Southwest Hospital, Third Military Medical University, Chongqing 400038, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No. 30070348
Correspondence to: Professor Jian-Min Yang, Gastroenterology Research Center, Southwest Hospital, Third Military Medical University, Chongqing 400038, China. jianminyang@hotmail.com
Telephone: +86-23-68754678 Fax: +86-537-2903067
Received: August 6, 2003
Revised: September 18, 2003
Accepted: October 12, 2003
Published online: July 15, 2004
Abstract

AIM: To study the effects of sense and antisense KAI1 genes on the growth and invasion of human hepatocellular carcinoma (HCC) cell line MHCC97-H.

METHODS: KAI1 sense and antisense eukaryotic expression plasmids were constructed using subclone technique and transfected into MHCC97-H cells respectively by DOTAP liposome. After successful transfection was confirmed, in vitro growth curve, cell cycles, plate clone formation efficiency, invasive ability in Boyden Chamber assay and ultrastructural morphology were studied.

RESULTS: KAI1 sense and antisense genes had no significant effects on the cell growth curve and cell cycles. After transfection with sense KAI1 gene, decreased invasive ability in Boyden Chamber assay and decreased amount of mitochondria, but no significant changes of plate clone formation efficiency were observed in MHCC97-H-S cells. The plate clone formation efficiency and invasive ability in Boyden Chamber assay were significantly increased in MHCC97-H-AS cells, after transfection with antisense KAI1 gene. Furthermore, increased amount of mitochondria, rough endoplasmic reticulum, Golgi apparatus and expanded endoplasmic reticulum were also noted in MHCC97-H-AS cells.

CONCLUSION: Changes of KAI1 expression in HCC cells may alter their invasive and metastasis ability of the tumor.

Keywords: $[Keywords]