Brief Reports
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jul 1, 2004; 10(13): 1958-1960
Published online Jul 1, 2004. doi: 10.3748/wjg.v10.i13.1958
Comparison of three PCR methods for detection of Helicobacter pylori DNA and detection of cagA gene in gastric biopsy specimens
SI Smith, KS Oyedeji, AO Arigbabu, F Cantet, F Megraud, OO Ojo, AO Uwaifo, JA Otegbayo, SO Ola, AO Coker
SI Smith, Molecular Biology and Biotechnology Division, Nigerian Institute of Medical Research, P.M.B. 2013, Yaba, Lagos, Nigeria
KS Oyedeji, Microbiology Division, Nigerian Institute of Medical Research, P.M.B. 2013, Yaba, Lagos, Nigeria
AO Arigbabu, Department of Surgery, Obafemi Awolowo University Teaching Hospital Complex, Ile-Ife
F Cantet, F Megraud, Laboratoire de Bacteriologie, Bordeaux, Cedex, France
OO Ojo, AO Uwaifo, Department of Biochemistry, University of Ibadan, Ibadan, Nigeria
JA Otegbayo, SO Ola, Department of Medicine, University of Ibadan, Ibadan, Nigeria
AO Coker, College of Medicine, University of Lagos, Idi-Araba, Lagos, Nigeria
Author contributions: All authors contributed equally to the work.
Supported by Inserm Fellowship, France, awarded to Dr. SI Smith
Correspondence to: Dr. SI Smith, Molecular Biology and Biotechnology Division, Nigerian Institute of Medical Research, P.M.B. 2013, Yaba, Lagos, Nigeria. stellaismith@yahoo.com
Fax: +2341-342-5171
Received: January 15, 2004
Revised: April 7, 2004
Accepted: April 14, 2004
Published online: July 1, 2004
Abstract

AIM: To comparatively evaluate PCR and other diagnostic methods (the rapid urease test and/or culture) in order to determine which of the three PCR methods (ureA, glmM and 26-kDa, SSA gene) was most appropriate in the diagnosis of Helicobacter pylori (H pylori ) infection and also to evaluate the detection of a putative virulence marker of H pylori, the cagA gene, by PCR in biopsy specimens.

METHODS: One hundred and eighty-nine biopsy specimens were collected from 63 patients (three biopsies each) undergoing upper gastroduodenal endoscopy for various dyspeptic symptoms. The PCR methods used to detect H pylori DNA directly from biopsies were the glmM, 26-kDa, ureA and then cagA was used to compare the culture technique and CLO for urease with the culture technique being used as the gold standard.

RESULTS: Thirty-five percent of the biopsies were positive for H pylori DNA using the 3 PCR methods, while 68% of these were positive for the cagA gene. Twenty-four percent of the biopsies were negative for H pylori DNA in all PCR methods screened. The remaining 41% were either positive for ureA gene only, glmM only, 26-kDa only, or ureA + glmM, ureA + 26-kDa, glmM + 26-kDa. Out of the 35% positive biopsies, 41% and 82% were positive by culture and CLO respectively, while all negative biopsies were also negative by culture and cagA. Cag A+ infection was also predominantly found in H pylori DNA of the biopsies irrespective of the clinical diagnosis.

CONCLUSION: This method is useful for correctly identifying infections caused by H pylori and can be easily applied in our laboratory for diagnostic purposes.

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