Case Report
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jun 1, 2004; 10(11): 1686-1687
Published online Jun 1, 2004. doi: 10.3748/wjg.v10.i11.1686
Fatal liver failure due to reactivation of lamivudine-resistant HBV mutant
Tatehiro Kagawa, Norihito Watanabe, Hisashi Kanouda, Ichiro Takayama, Tadahiko Shiba, Takashi Kanai, Kazuya Kawazoe, Shinji Takashimizu, Nobue Kumaki, Kazuo Shimamura, Shohei Matsuzaki, Tetsuya Mine
Tatehiro Kagawa, Norihito Watanabe, Hisashi Kanouda, Ichiro Takayama, Tadahiko Shiba, Takashi Kanai, Kazuya Kawazoe, Shinji Takashimizu, Shohei Matsuzaki, Tetsuya Mine, Department of Internal Medicine, Tokai University School of Medicine, Boseidai, Isehara, Kanagawa 259-1193, Japan
Nobue Kumaki, Kazuo Shimamura, Department of Pathology, Tokai University School of Medicine, Boseidai, Isehara, Kanagawa 259-1193, Japan
Author contributions: All authors contributed equally to the work.
Correspondence to: Dr. Tatehiro Kagawa, Department of Internal Medicine, Tokai University School of Medicine, Boseidai, Isehara, Kanagawa 259-1193, Japan. kagawa@is.icc.u-tokai.ac.jp
Telephone: +81-463-931121
Received: November 17, 2003
Revised: February 4, 2004
Accepted: February 11, 2004
Published online: June 1, 2004
Abstract

We report a case of fatal liver failure due to reactivation of lamivudine-resistant HBV. A 53-year-old man was followed since 1998 for HBV-related chronic hepatitis. Serum HBV-DNA was 150 MEq/mL (branched DNA signal amplification assay) and ALT levels fluctuated between 50-200 IU/L with no clinical signs of liver cirrhosis. Lamivudine (100 mg/d) was started in May 2001 and serum HBV-DNA subsequently decreased below undetectable levels. In May 2002, serum HBV-DNA had increased to 410 MEq/mL, along with ALT flare (226 IU/L). The YMDD motif in the DNA polymerase gene had been replaced by YIDD. Lamivudine was continued and ALT spontaneously decreased to the former levels. On Oct 3 the patient presenting with general fatigue, nausea and jaundice was admitted to our hospital. The laboratory data revealed HBV reactivation and liver failure (ALT: 1828 IU/L, total bilirubin: 10 mg/dL, and prothrombin INR: 3.24). For religious reasons, the patient and his family refused blood transfusion, plasma exchange and liver transplantation. The patient died 10 d after admission. The autopsy revealed remarkable liver atrophy.

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