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World J Transplant. Jun 18, 2026; 16(2): 118288
Published online Jun 18, 2026. doi: 10.5500/wjt.v16.i2.118288
Figure 1
Figure 1 Life-course framework of cardiovascular risk after liver transplantation. CV: Cardiovascular.
Figure 2
Figure 2 Linking immunosuppressive injury pathways to noninvasive vascular phenotypes. NO: Nitric oxide; eNOS: Endothelial nitric oxide synthase; PWV: Pulse wave velocity; AIx: Augmentation index; cIMT: Carotid intima-media thickness; FMD: Flow-mediated dilation; CT calcium: Coronary artery calcium score; CNI: Calcineurin inhibitor; CKD: Chronic kidney disease.
Figure 3
Figure 3 Comparative vascular and metabolic profiles of major immunosuppressive classes. mTOR: Mammalian target of rapamycin; VEGF: Vascular endothelial growth factor.
Figure 4
Figure 4 Comparative mechanistic burden across drug classes. NO: Nitric oxide; CNI: Calcineurin inhibitor; mTORi: Mammalian target of rapamycin inhibitor; CV: Cardiovascular; mTOR: Mammalian target of rapamycin.


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