Attenuating neuropsychiatric disorders of early-life stress: The protective role of oxytocin

doi:10.5498/wjp.v16.i4.115109

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Apr 19, 2026 (publication date) through Mar 30, 2026

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World Journal of Psychiatry

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2220-3206

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Psychiatry. Apr 19, 2026; 16(4): 115109
Published online Apr 19, 2026. doi: 10.5498/wjp.v16.i4.115109

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Figure 1 Neurobiological sequelae of early-life stress. ELS: Early-life stress; HPA: Hypothalamic-pituitary-adrenal; CRF: Corticotropin-releasing factor; ACTH: Adrenocorticotropic hormone.

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Figure 2 Multi-level protective mechanisms of oxytocin intervention. GR: Glucocorticoid receptor; HPA: Hypothalamic-pituitary-adrenal; BDNF: Brain-derived neurotrophic factor; CRF: Corticotropin-releasing factor; ACTH: Adrenocorticotropic hormone.

Citation: Zhang Y, Wang S, Hei MY. Attenuating neuropsychiatric disorders of early-life stress: The protective role of oxytocin. World J Psychiatry 2026; 16(4): 115109
URL: https://www.wjgnet.com/2220-3206/full/v16/i4/115109.htm
DOI: https://dx.doi.org/10.5498/wjp.v16.i4.115109