Published online Feb 23, 2016. doi: 10.5494/wjh.v6.i1.60
Peer-review started: August 3, 2015
First decision: September 29, 2015
Revised: October 30, 2015
Accepted: December 29, 2015
Article in press: December 31, 2015
Published online: February 23, 2016
Processing time: 205 Days and 0.7 Hours
Renal venous hypertension usually seen in young, otherwise healthy individuals and can lead to significant overall morbidity. Aside from clinical findings and physical examination, diagnosis can be made with ultrasound, computed tomography, or magnetic resonance conventional venography. Symptoms and haemodynamic significance of the compression determine the ideal treatment method. This review of the literature discusses normal and pathological developmental aspects of renocaval venous segment and related circulatory disorders, summarizes congenital and acquired changes in left renal vein and their impact on development of renal venous hypertension. Also will be discussed surgical tactics of portosystemic shunting and their potential effects on renal hemodynamics.
Core tip: Renal venous hypertension characterized by the presence of left renal vein dilatation, varicocele and hematuria. Being a rare cause of hematuria its etiology is diverse but of precise characteristics. Diagnosis is not easy and treatment requires ruling out its precise etiology and considering the intensity of the compression phenomenon because of interventionist attitudes have important implications and are not risk free.