Published online Feb 8, 2016. doi: 10.5409/wjcp.v5.i1.82
Peer-review started: July 15, 2015
First decision: September 17, 2015
Revised: November 9, 2015
Accepted: November 17, 2015
Article in press: November 25, 2015
Published online: February 8, 2016
Processing time: 197 Days and 20.9 Hours
Helicobacter pylori (H. pylori) is one of the factors involved in the pathogenesis of various gastrointestinal diseases and may play a potential role in certain extra-intestinal diseases. H. pylori infection are mainly acquired during childhood, and it has been reported that in endemic areas of China the infection rates are extraordinarily higher in HSP children, particular those with abdominal manifestations. Furthermore, eradication therapy may ameliorate Henoch-Schonlein purpura (HSP) manifestations and decrease the recurrence of HSP. Therefore, results suggested that detection of H. pylori infection by appropriate method ought to be applied in HSP children. Current evidences indicate that local injury of gastric mucosa and immunological events induced by H. pylori infection are involved in the development of HSP. Increased serum IgA, cryoglobulins, C3 levels, autoimmunity, proinflammatory substances and molecular mimicry inducing immune complex and cross-reactive antibodies caused by H. pylori infection might play their roles in the course of HSP. However, there are no investigations confirming the causality between H. pylori infection and HSP, and the pathogenesis mechanism is still unclear. More bench and clinical studies need to be executed to elaborate the complex association between H. pylori and HSP.
Core tip: This is the first comprehensive review to report current clinical and bench studies focusing on the potential role of Helicobacter pylori infection in Henoch-Schonlein purpura children. We also presented the possible mechanism underlying their association and the questions need to be addressed in the future studies.