Mechanisms involved in Helicobacter pylori induced duodenal ulcer disease: an overview

doi:10.3748/wjg.v6.i5.619

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Oct 15, 2000 (publication date) through Nov 6, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 15, 2000; 6(5): 619-623
Published online Oct 15, 2000. doi: 10.3748/wjg.v6.i5.619

Table 1 Gastric acid secretory characteristics of duodenal patients (all of which might be Helicobacter pylori induced)

Increased maximal acid secretory capacity

Increased parietal cell mass (trophic effect of gastrin?)

Increased basal acid secretion

Increased basal release of gastrin

Increased and prolonged acid response to meals

Increased release of gastrin

Defective inhibitory mechanisms

Antral acidification

Antral distension

Fat in the duodenum (H. pylori induced?)

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Citation: Olbe L, Fändriks L, Hamlet A, Svennerholm AM, Thoreson AC. Mechanisms involved in Helicobacter pylori induced duodenal ulcer disease: an overview. World J Gastroenterol 2000; 6(5): 619-623
URL: https://www.wjgnet.com/1007-9327/full/v6/i5/619.htm
DOI: https://dx.doi.org/10.3748/wjg.v6.i5.619