©The Author(s) 1998.
World J Gastroenterol. Oct 15, 1998; 4(Suppl2): 38-40
Published online Oct 15, 1998. doi: 10.3748/wjg.v4.iSuppl2.38
Published online Oct 15, 1998. doi: 10.3748/wjg.v4.iSuppl2.38
Table 1 Histopathological changes
| Gallbladder mucosa | Proximal end of cystic duct | Distant end of cystic duct | Duodenum | Neighbouring liver tissues | |
| No.1 (2 wk) | Coagulative necrosis, focal chronic inflammatory reaction, a great deal of inflammatory granulation tissues | Mucosal necrosis, chronic inflammatory reaction | Mucosa present, chronic inflammatory reaction | No change | No change |
| No.2 (4 wk) | Complete necrosis, chronic inflammatory reaction, granulation tissue and scar formation | Mucosal necrosis, chronic inflammation | Slight chronic inflammatory reaction | No change | No change |
| No.3 (6 wk) | Complete necrosis, replaced by inflammatory granulation tissue and scar formation | Mucosal destruction, granulation tissues | Chronic inflammatory reation | No change | No change |
| No.4 (8 wk) | Mucosal coagulative necrosis, inflammatory granulation tissue and scar formation | Mucosal destruction, fibrous connective tissue | Inflamm atory reaction | No change | No change |
| No.5 (10 wk) | Mucosa completely destroyed, dropping out, scar formation | Mucosal destruction, fibrous connective tissue | No pathologic change | No change | No change |
- Citation: Sun SM, Xu JH, Sun SQ, Ma T, Wu MY, Wu LB, Chen K, Liu WX. Experimental study of chemical cholecystectomy through abdominoscopic technology. World J Gastroenterol 1998; 4(Suppl2): 38-40
- URL: https://www.wjgnet.com/1007-9327/full/v4/iSuppl2/38.htm
- DOI: https://dx.doi.org/10.3748/wjg.v4.iSuppl2.38
