Iron and non-alcoholic fatty liver disease

doi:10.3748/wjg.v22.i36.8112

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Sep 28, 2016 (publication date) through Nov 12, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Sep 28, 2016; 22(36): 8112-8122
Published online Sep 28, 2016. doi: 10.3748/wjg.v22.i36.8112

Table 1 Proposed mechanisms for the involvement of iron in non-alcoholic fatty liver disease pathogenesis

Site	Mechanism
Hepatic iron	Oxidative Stress[31,53-57]
	Reduced VLDL export[51]
	Macrophage activation[57-59]
	Stellate cell activation[60]
	Endoplasmic reticulum stress[61]
	Increased cholesterol synthesis[62]
Adipose tissue iron	Reduced adiponectin[19,73,74]
	Reduced leptin[76]
	Increased resistin[75]
	Increased lipolysis[77,78]

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Citation: Britton LJ, Subramaniam VN, Crawford DH. Iron and non-alcoholic fatty liver disease. World J Gastroenterol 2016; 22(36): 8112-8122
URL: https://www.wjgnet.com/1007-9327/full/v22/i36/8112.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i36.8112