Inflammatory microenvironment contributes to epithelial-mesenchymal transition in gastric cancer

doi:10.3748/wjg.v22.i29.6619

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Aug 7, 2016; 22(29): 6619-6628
Published online Aug 7, 2016. doi: 10.3748/wjg.v22.i29.6619

Table 1 Important inflammatory mediators involved in the EMT in GC

Categories	Factors	Ref
Cytokines	TGF-β1	[103,104]
	TNF-α	[99,100]
	TGF-α	[108]
	IL-6	[109,110]
	IL-8	[102]
Chemokines and receptors	CCL5	[84,131]
	CCL18	[132,133]
	CCR2	[134]
	CCR7	[135]
	CCL20-CCR6	[136,137]
	CXCR1	[138,139]
	CXCR3	[140]
	CXCL12-CXCR4	[108,117]
Immune cells	CD8⁺ cytotoxic T cells	[56,60]
	Th1/Th2 cells ratio	[63]
	Th17 cells	[64,65,67]
	Foxp3⁺ Tregs	[68-70]
	Foxp3⁺/CD4⁺ ratio	[71]
	Foxp3⁺/CD8⁺ ratio	[68]
	NK cells	[87-89]
	TAMs	[78,79]
	DCs	[93-95]
	TAM/Foxp3⁺ ratio	[141]
MMPs	MMP-2	[125]
	MMP-9	[124-126,142]
	MMP-7	[51,127,128]

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Citation: Ma HY, Liu XZ, Liang CM. Inflammatory microenvironment contributes to epithelial-mesenchymal transition in gastric cancer. World J Gastroenterol 2016; 22(29): 6619-6628
URL: https://www.wjgnet.com/1007-9327/full/v22/i29/6619.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i29.6619