Copyright
©The Author(s) 2000.
World J Gastroenterol. Aug 15, 2000; 6(4): 540-545
Published online Aug 15, 2000. doi: 10.3748/wjg.v6.i4.540
Published online Aug 15, 2000. doi: 10.3748/wjg.v6.i4.540
Figure 1 Collagen expression in liver section of model rat (van Gieson staining, × 200) (A), and of losartan treated group (van Giesion staining, × 200).
Significantly less fibrosis can be noted compared to that in the model group (B).
Figure 2 AT1 receptor expression in liver section of model rat (DAB staining, × 200).
AT1 receptor is seen mainly scattered in fibrotic areas and vascular wal l (A), and of los artan treated group (DAB staining, × 200). Note significantly less AT1 expression than that seen in the model group (B).
Figure 3 TGF-β expression in liver section of model rat ( DAB staining, × 200).
TGF-β expression is seen scattered in the fibrotic areas and vascular wall (A), and of losart an treated group (DAB staining, × 200). Note significantly less TGF-β expression than that seen in the model group (B).
- Citation: Wei HS, Li DG, Lu HM, Zhan YT, Wang ZR, Huang X, Zhang J, Cheng JL, Xu QF. Effects of AT1 receptor antagonist, losartan, on rat hepatic fibrosis induced by CCl4. World J Gastroenterol 2000; 6(4): 540-545
- URL: https://www.wjgnet.com/1007-9327/full/v6/i4/540.htm
- DOI: https://dx.doi.org/10.3748/wjg.v6.i4.540