Co-regulative effects of the cAMP/PKA and DAG/PKC signal pathways on human gastric cancer cells during differentiation induced by traditional Chinese medicines

Figure 1 Changes of cAMP level in MGc80-3 cells treated in different conditions. A: control; B: 1.8 g/L bailong; C: 1.8 g/L bailong + 20 mg/L PKA inhibitor; D: 5 mmol/L PKC inhibitor (n = 3)

Figure 2 Changes of PKA activity in MGc80-3 cells treated in different conditions. A: control; B: 1.8 g/L bailong; C: 1.8 g/L bailing + 20 mg/L PKA inhibitor; D: 5 mmol/L PKC inhibitor (n = 3)

Figure 3 Changes of DAG activity in MGc80-3 cells treated in different conditions. A: control; B: 1.8 mg/L bailong; C: 1.8 g/L bailing + 20 mg/L PKA inhibitor; D: 5 mmol/L (n = 3)

Figure 4 Changes of PKC activity in MGc80-3 cells treated in different conditions. A: control; B: 1.8 g/L bailong; C: 1.8 g/L bailing + 20 mg/L PKA inhibitor; D: 5 mmol/L PKC inhibitor (n = 3)