Mechanisms of triglyceride metabolism in patients with bile acid diarrhea

doi:10.3748/wjg.v22.i30.6757

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Aug 14, 2016; 22(30): 6757-6763
Published online Aug 14, 2016. doi: 10.3748/wjg.v22.i30.6757

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Figure 1 Relationship of triglycerides to selenium-75-homophoric acid taurine retention values in patients with primary and secondary bile acid diarrhoea. The linear regression (R² value 0.0129) and normal values for TGs (1.9 mmol/L) and selenium-75-homophoric acid taurine (SeHCAT) retention (15%) are shown.

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Figure 2 Overview of the farnesoid X receptor action on bile acid synthesis and triglyceride metabolism. SHP: Small heterodimer primer; BA: Bile acid; FGF: Fibroblast growth factor; FFA: Free fatty acid; LPL: Lipoprotein lipase; VLDL: Very low-density lipoproteins; CR: Chylomicron; ASBT: Apical ileal sodium dependant bile acid transporter; MTP: Microsomal triglyceride transfer protein; PPAR: peroxisome proliferator-activated receptor; SREBP: Sterol regulatory element binding protein; FXR: Farnesoid X receptor.

Citation: Sagar NM, McFarlane M, Nwokolo C, Bardhan KD, Arasaradnam RP. Mechanisms of triglyceride metabolism in patients with bile acid diarrhea. World J Gastroenterol 2016; 22(30): 6757-6763
URL: https://www.wjgnet.com/1007-9327/full/v22/i30/6757.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i30.6757