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Aug 14, 2015 (publication date) through Apr 29, 2026
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World Journal of Gastroenterology
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1007-9327
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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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©The Author(s) 2015.
World J Gastroenterol. Aug 14, 2015; 21(30): 9021-9037
Published online Aug 14, 2015. doi: 10.3748/wjg.v21.i30.9021
Figure 1
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Figure 1 Proposed mechanism by which Helicobacter pylori induced epigenetic alteration affecting DNA repair. The more virulent Helicobacter pylori strains (cagA and vacA s1m1) increase the inflammatory response, 8-oxodG and ROS production on epithelial cells. This landscape may cause the hypermethylation of CpG islands and/or the histone hypoacetylation, which results the in the DNA repair gene silencing. It might increase the mutation rate which may contribute to the genomic instability and gastric cancer development.
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