Exogenous acid fibroblast growth factor inhibits ischemia-reperfusion-induced damage in intestinal epithelium via regulating P53 and P21WAF-1 expression

doi:10.3748/wjg.v11.i44.6981

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Nov 28, 2005 (publication date) through Nov 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 28, 2005; 11(44): 6981-6987
Published online Nov 28, 2005. doi: 10.3748/wjg.v11.i44.6981

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Figure 1 Expression of P53 and P21^WAF-1 protein in C group (A and D, respectively), A group (B and E, respectively) and R group (C and F, respectively).

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Figure 2 Expression of P53, P21^WAF-1, and β-actin genes in normal saline-treated (A, C, E, respectively) and aFGF treated (B, D, F, respectively) rat intestinal villi. Bar indicates the size of RT-PCR cDNA products. Lanes 1-8: 48, 24, 12, 6, 2, 1 h, and 30, 15 min after reperfusion; lane 9: ischemia group; lane 10: sham-operated control group; lane 11: DL 2000 marker.

Citation: Chen W, Fu XB, Ge SL, Li WJ, Sun TZ, Sheng ZY. Exogenous acid fibroblast growth factor inhibits ischemia-reperfusion-induced damage in intestinal epithelium via regulating P53 and P21WAF-1 expression. World J Gastroenterol 2005; 11(44): 6981-6987
URL: https://www.wjgnet.com/1007-9327/full/v11/i44/6981.htm
DOI: https://dx.doi.org/10.3748/wjg.v11.i44.6981