Copyright
©The Author(s) 2005.
World J Gastroenterol. Aug 7, 2005; 11(29): 4451-4456
Published online Aug 7, 2005. doi: 10.3748/wjg.v11.i29.4451
Published online Aug 7, 2005. doi: 10.3748/wjg.v11.i29.4451
Figure 1 Cisplatin and DTT treatments induce DNA ladder formation in Eca109 cells.
Figure 2 Activation of P38 kinase in DTT-treated Eca109 cells.
Figure 3 Activation of P38 kinase in cisplatin-treated Eca109 cells.
Figure 4 Activation of P38 kinase in DTT- and cisplatin-treated Eca109 cells.
Figure 5 Activation of P38 kinase in untreated Eca109 cells.
Figure 6 Negative control.
Figure 7 The effects of SB203580 on DTT- and cisplatin-induced Eca109 cell apoptosis – flow cytometry analysis.
SB sharply decreased the apoptotic peak of the group treated with DTT or cisplatin or DTT plus cisplatin. D: DTT, Di: DTT-SB203580, C: cisplatin, Ci: cisplatin-SB203580, D+C: DTT+cisplatin, D+Ci: DTT+cisplatin-SB203580, U: untreated, a: apoptotic peak.
- Citation: Zhang QX, Feng R, Zhang W, Ding Y, Yang JY, Liu GH. Role of stress-activated MAP kinase P38 in cisplatin- and DTT-induced apoptosis of the esophageal carcinoma cell line Eca109. World J Gastroenterol 2005; 11(29): 4451-4456
- URL: https://www.wjgnet.com/1007-9327/full/v11/i29/4451.htm
- DOI: https://dx.doi.org/10.3748/wjg.v11.i29.4451