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World J Gastroenterol. Feb 7, 2012; 18(5): 412-424
Published online Feb 7, 2012. doi: 10.3748/wjg.v18.i5.412
Published online Feb 7, 2012. doi: 10.3748/wjg.v18.i5.412
Role of ATG16L, NOD2 and IL23R in Crohn’s disease pathogenesis
Saleh A Naser, Melissa Arce, Anam Khaja, Marlene Fernandez, Sammer Elwasila, Saisathya Thanigachalam, Department of Molecular Biology and Microbiology, Burnett School of Biomedical Sciences, College of Medicine, University of Central Florida, Orlando, FL 32816, United States
Najih Naser, Division of Research and Development, BioSynse, Cary, NC 27513, United States
Author contributions: All authors made substantial contributions to conception and design, drafting the article and revising it critically for important intellectual content; and all authors approved the version to be published.
Supported by The Broad Foundation grant, No. IBD-0207R
Correspondence to: Saleh A Naser, PhD, Professor, Graduate, Coordinator, Department of Molecular Biology and Microbiology, Burnett School of Biomedical Sciences, College of Medicine, University of Central Florida, Orlando, FL 32816, United States. nasers@mail.ucf.edu
Telephone: +1-407-8230955 Fax: +1-407-8230956
Received: April 1, 2011
Revised: August 8, 2011
Accepted: August 27, 2011
Published online: February 7, 2012
Revised: August 8, 2011
Accepted: August 27, 2011
Published online: February 7, 2012
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