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Cited by in CrossRef
For: Wolfkamp SC, Verseyden C, Vogels EW, Meisner S, Boonstra K, Peters CP, Stokkers PC, te Velde AA. ATG16L1 and NOD2 polymorphisms enhance phagocytosis in monocytes of Crohn’s disease patients. World J Gastroenterol 2014; 20(10): 2664-2672 [PMID: 24627602 DOI: 10.3748/wjg.v20.i10.2664]
URL: https://www.wjgnet.com/1007-9327/full/v20/i10/2664.htm
Number Citing Articles
1
Yuqing Wang, Jinglan Nie, Guowang Lin, Xiaolong He, Hongwei Zhou. Genetic and microbial regulation of NOD2 signaling in homeostasis and diseaseMedScience 2026;  doi: 10.1007/s11684-026-1239-6
2
Di Wu, Tomoko Matsuda, Danielle Xie, Min Shen, Atika Dhar, John M. Davis, Bo Shen, Naotomo Kambe, Warren Strober, Qingping Yao. NOD2-Related Multisystem Inflammatory Disorders and Recent AdvancesCurrent Rheumatology Reports 2026; 28(1) doi: 10.1007/s11926-026-01212-4
3
Mark S. Gresnigt, Cristina Cunha, Martin Jaeger, Samuel M. Gonçalves, R. K. Subbarao Malireddi, Anne Ammerdorffer, Rosalie Lubbers, Marije Oosting, Orhan Rasid, Grégory Jouvion, Catherine Fitting, Dirk J. de Jong, João F. Lacerda, António Campos, Willem J. G. Melchers, Katrien Lagrou, Johan Maertens, Thirumala-Devi Kanneganti, Agostinho Carvalho, Oumaima Ibrahim-Granet, Frank L. van de Veerdonk. Genetic deficiency of NOD2 confers resistance to invasive aspergillosisNature Communications 2018; 9(1) doi: 10.1038/s41467-018-04912-3
4
Virginie Mercier, Gabrielle Boucher, Dominic Devost, Kyla Bourque, Azadeh Alikashani, Claudine Beauchamp, Alain Bitton, Sylvain Foisy, Philippe Goyette, Guy Charron, Terence E. Hébert, John D. Rioux. IBD-associated G protein-coupled receptor 65 variant compromises signalling and impairs key functions involved in inflammationCellular Signalling 2022; 93 doi: 10.1016/j.cellsig.2022.110294
5
Xiping Liao, Ji Liu, Xiaolong Guo, Ruiping Meng, Wei Zhang, Jianyun Zhou, Xia Xie, Hongli Zhou. Origin and Function of Monocytes in Inflammatory Bowel DiseaseJournal of Inflammation Research 2024;  doi: 10.2147/JIR.S450801
6
Dana Elshaer, Jakob Begun. The role of barrier function, autophagy, and cytokines in maintaining intestinal homeostasisSeminars in Cell & Developmental Biology 2017; 61 doi: 10.1016/j.semcdb.2016.08.018
7
Mohammad Salem, Mette Ammitzboell, Kris Nys, Jakob Benedict Seidelin, Ole Haagen Nielsen. ATG16L1: A multifunctional susceptibility factor in Crohn diseaseAutophagy 2015; 11(4) doi: 10.1080/15548627.2015.1017187
8
Mayada Mazher, Yomna Adel Moqidem, Mona Zidan, Ahmed A. Sayed, Ahmed Abdellatif. Autophagic reprogramming of bone marrow–derived macrophagesImmunologic Research 2023; 71(2) doi: 10.1007/s12026-022-09344-2
9
Martina Girardelli, Federica Basaldella, Sara Della Paolera, Josef Vuch, Alberto Tommasini, Stefano Martelossi, Sergio Crovella, Anna Monica Bianco. Genetic profile of patients with early onset inflammatory bowel diseaseGene 2018; 645 doi: 10.1016/j.gene.2017.12.029
10
Ebrahim Alijani, Mohammad Naderi, Behrouz Mollashahi, Mahdi Atabaki. Association between NOD2 gene polymorphisms and susceptibility to pulmonary tuberculosis in Zahedan, Southeast IranGene Reports 2021; 25 doi: 10.1016/j.genrep.2021.101395
11
Claire L. Feerick, Declan P. McKernan. Understanding the regulation of pattern recognition receptors in inflammatory diseases – a ‘Nod’ in the right directionImmunology 2017; 150(3) doi: 10.1111/imm.12677
12
Qi Wang, Qingfa Bu, Zibo Xu, Yuan Liang, Jinren Zhou, Yufeng Pan, Haoming Zhou, Ling Lu. Macrophage ATG16L1 expression suppresses metabolic dysfunction-associated steatohepatitis progression by promoting lipophagyClinical and Molecular Hepatology 2024; 30(3) doi: 10.3350/cmh.2024.0107
13
Ferenc Sipos, Györgyi Műzes. Autophagy Networks in Inflammation2016;  doi: 10.1007/978-3-319-30079-5_12
14
Hujun Cui, Yunlong Liu, Yancui Zheng, Hongping Li, Minying Zhang, Xu Wang, Xiaoheng Zhao, Hanliang Cheng, Jianhe Xu, Xiangning Chen, Zhujin Ding. Intelectin enhances the phagocytosis of macrophages via CDC42-WASF2-ARPC2 signaling axis in Megalobrama amblycephalaInternational Journal of Biological Macromolecules 2023; 236 doi: 10.1016/j.ijbiomac.2023.124027
15
Mauricio Javier Olivares-Morales, Marjorie Katherine De La Fuente, Karen Dubois-Camacho, Daniela Parada, David Diaz-Jiménez, Alejandro Torres-Riquelme, Xiaojiang Xu, Nayaret Chamorro-Veloso, Rodrigo Naves, Maria-Julieta Gonzalez, Rodrigo Quera, Carolina Figueroa, John Anthony Cidlowski, Roberto Mauricio Vidal, Marcela Alejandra Hermoso. Glucocorticoids Impair Phagocytosis and Inflammatory Response Against Crohn’s Disease-Associated Adherent-Invasive Escherichia coliFrontiers in Immunology 2018; 9 doi: 10.3389/fimmu.2018.01026
16
Shahanavaj Khan, Ahamad Imran, Abdul Malik, Anis Ahmad Chaudhary, Abdur Rub, Arif Tasleem Jan, Jakeera Begum Syed, Christian Rolfo. Bacterial imbalance and gut pathologies: Association and contribution of E. coli in inflammatory bowel disease Critical Reviews in Clinical Laboratory Sciences 2019; 56(1) doi: 10.1080/10408363.2018.1517144
17
M.E. Wildenberg, P.J. Koelink, K. Diederen, A.A. te Velde, S C S Wolfkamp, V.J. Nuij, M.P. Peppelenbosch, M. Nobis, O.J. Sansom, K.I. Anderson, C.J. van der Woude, G R A M D'Haens, G.R. van den Brink. The ATG16L1 risk allele associated with Crohn's disease results in a Rac1-dependent defect in dendritic cell migration that is corrected by thiopurinesMucosal Immunology 2017; 10(2) doi: 10.1038/mi.2016.65
18
Kirsty M Hooper, Peter G Barlow, Paul Henderson, Craig Stevens. Interactions Between Autophagy and the Unfolded Protein Response: Implications for Inflammatory Bowel DiseaseInflammatory Bowel Diseases 2019; 25(4) doi: 10.1093/ibd/izy380
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