| For: | Bene J, Magyari L, Talián G, Komlósi K, Gasztonyi B, Tari B, Várkonyi Á, Mózsik G, Melegh B. Prevalence of SLC22A4, SLC22A5 and CARD15 gene mutations in Hungarian pediatric patients with Crohn’s disease. World J Gastroenterol 2006; 12(34): 5550-5553 [PMID: 17006998 DOI: 10.3748/wjg.v12.i34.5550] |
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| URL: | https://www.wjgnet.com/1007-9327/full/v12/i34/5550.htm |
| Number | Citing Articles |
| 1 |
Anton G. Kutikhin, Arseniy E. Yuzhalin. Genomics of Pattern Recognition Receptors. 2013; doi: 10.1007/978-3-0348-0688-6_7
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| 2 |
Lilla Lakner, Veronika Csöngei, Patrícia Sarlós, Luca Járomi, Enikő Sáfrány, Márta Varga, Péter Orosz, Lili Magyari, Judit Bene, Pál Miheller, Zsolt Tulassay, Béla Melegh. IGR2096a_1 T and IGR2198a_1 C alleles on IBD5 locus of chromosome 5q31 region confer risk for Crohn’s disease in Hungarian patients. International Journal of Colorectal Disease 2009; 24(5) doi: 10.1007/s00384-009-0670-x
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| 3 |
Current World Literature. Current Opinion in Pediatrics 2007; 19(5) doi: 10.1097/MOP.0b013e3282f12851
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| 4 |
Jian Wang, Xi Wang, Hong Yang, Dong Wu, Li Wang, Jiaming Qian. Contribution of the IBD5 locus to inflammatory bowel disease: a meta-analysis. Human Genetics 2011; 129(6) doi: 10.1007/s00439-011-0952-6
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| 5 |
Gilaad G Kaplan, Bo V Pedersen, Roland E Andersson, Bruce E Sands, Joshua Korzenik, Morten Frisch. The risk of developing Crohn’s disease after an appendectomy: a population-based cohort study in Sweden and Denmark. Gut 2007; 56(10) doi: 10.1136/gut.2007.121467
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| 6 |
Takuya Shimizu, Yusuke Masuo, Saki Takahashi, Noritaka Nakamichi, Yukio Kato. Organic cation transporter Octn1-mediated uptake of food-derived antioxidant ergothioneine into infiltrating macrophages during intestinal inflammation in mice. Drug Metabolism and Pharmacokinetics 2015; 30(3) doi: 10.1016/j.dmpk.2015.02.003
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| 7 |
Károly Lapis. Barrier- and autophagic functions of the intestinal epithelia: role of disturbances in the pathogenesis of Crohn’s disease. Orvosi Hetilap 2010; 151(40) doi: 10.1556/oh.2010.28942
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| 8 |
Anton G. Kutikhin. Role of NOD1/CARD4 and NOD2/CARD15 gene polymorphisms in cancer etiology. Human Immunology 2011; 72(10) doi: 10.1016/j.humimm.2011.06.003
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| 9 |
Shiva Yazdanyar, Maren Weischer, Børge G Nordestgaard. Genotyping for NOD2 Genetic Variants and Crohn Disease: a Metaanalysis. Clinical Chemistry 2009; 55(11) doi: 10.1373/clinchem.2009.127126
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| 10 |
Prem S. Shekhawat, Srinivas Sonne, A. Lee Carter, Dietrich Matern, Vadivel Ganapathy. Enzymes involved in l-carnitine biosynthesis are expressed by small intestinal enterocytes in mice: Implications for gut health. Journal of Crohn's and Colitis 2013; 7(6) doi: 10.1016/j.crohns.2012.08.011
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| 11 |
Curtis D. Klaassen, Lauren M. Aleksunes. Xenobiotic, Bile Acid, and Cholesterol Transporters: Function and Regulation. Pharmacological Reviews 2010; 62(1) doi: 10.1124/pr.109.002014
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| 12 |
Johan Van Limbergen, Richard K. Russell, Elaine R. Nimmo, Jack Satsangi. The Genetics of Inflammatory Bowel Disease. The American Journal of Gastroenterology 2007; 102(12) doi: 10.1111/j.1572-0241.2007.01527.x
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| 13 |
G. Talián, L. Lakner, J. Bene, K. Komlósi, K. Horváth, B. Gasztonyi, P. Miheller, M. Figler, G. Mózsik, Z. Tulassay, B. Melegh. Plasma carnitine ester profiles in Crohn's disease and ulcerative colitis patients with different IGR2230a_1 genotypes. International Journal of Immunogenetics 2009; 36(6) doi: 10.1111/j.1744-313X.2009.00834.x
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| 14 |
Kek Heng CHUA, Lay Hoong LIAN, Boon Pin KEE, Chooi Mei THUM, Way Seah LEE, Ida HILMI, Khean Lee GOH. Identification of DLG5 and SLC22A5 gene polymorphisms in Malaysian patients with Crohn's disease. Journal of Digestive Diseases 2011; 12(6) doi: 10.1111/j.1751-2980.2011.00533.x
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| 15 |
Lili Magyari, Béla Melegh. Susceptibility genetic variants in Hungarian morbus Crohn and ulcerative colitis patients. Orvosi Hetilap 2009; 150(2) doi: 10.1556/oh.2009.28445
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| 16 |
Katja Repnik, Uroš Potočnik. Haplotype in the IBD5 region is associated with refractory Crohn’s disease in Slovenian patients and modulates expression of the SLC22A5 gene. Journal of Gastroenterology 2011; 46(9) doi: 10.1007/s00535-011-0426-6
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| 17 |
Prem S. Shekhawat, Sonne R. Srinivas, Dietrich Matern, Michael J. Bennett, Richard Boriack, Varghese George, Hongyan Xu, Puttur D. Prasad, Penny Roon, Vadivel Ganapathy. Spontaneous development of intestinal and colonic atrophy and inflammation in the carnitine-deficient jvs (OCTN2−/−) mice. Molecular Genetics and Metabolism 2007; 92(4) doi: 10.1016/j.ymgme.2007.08.002
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| 18 |
Lili Magyari, Judit Bene, Katalin Komlósi, Gábor Talián, Bernadett Faragó, Veronika Csöngei, Luca Járomi, Enikő Sáfrány, Csilla Sipeky, Lilla Lakner, Márta Varga, Beáta Gasztonyi, Béla Melegh. Prevalence of SLC22A4 1672T and SLC22A5 −207C combination defined TC haplotype in Hungarian ulcerative colitis patients. Pathology & Oncology Research 2007; 13(1) doi: 10.1007/BF02893441
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| 19 |
Lilla Lakner, Veronika Csöngei, Lili Magyari, Márta Varga, Pál Miheller, Patrícia Sarlós, Péter Orosz, Zsolt Bári, István Takács, Luca Járomi, Enikő Sáfrány, Csilla Sipeky, Judit Bene, Zsolt Tulassay, Zoltán Döbrönte, Béla Melegh. Possible role of selected IGR and SLC22A4/SLC22A5 loci in development of inflammatory bowel diseases. Orvosi Hetilap 2009; 150(29) doi: 10.1556/oh.2009.28677
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| 20 |
Yaru Xue, Chenhui Ma, Imad Hanna, Guoyu Pan. Drug Transporters in Drug Disposition, Effects and Toxicity. Advances in Experimental Medicine and Biology 2019; 1141 doi: 10.1007/978-981-13-7647-4_8
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| 21 |
Sarah M. AlQahtani, Reema A. Almutairi, Reem D. Al-Qahtani, Eman A. BinAqeel, Chittibabu Vatte, Cyril Cyrus. Association of SLC22A4 and TNF-α gene polymorphism with susceptibility to inflammatory bowel disease among patients in the Eastern Province of Saudi Arabia. Arab Journal of Gastroenterology 2025; 26(4) doi: 10.1016/j.ajg.2025.09.007
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| 22 |
Srinivas Sonne, Prem S. Shekhawat, Dietrich Matern, Vadivel Ganapathy, Leszek Ignatowicz, Markus M. Heimesaat. Carnitine Deficiency in OCTN2−/− Newborn Mice Leads to a Severe Gut and Immune Phenotype with Widespread Atrophy, Apoptosis and a Pro-Inflammatory Response. PLoS ONE 2012; 7(10) doi: 10.1371/journal.pone.0047729
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