Melatonin supplementation, hyperprolactinemia, and incident heart failure: A proposed prolactin-mediated pathway for cardiovascular risk

doi:10.5493/wjem.v16.i2.120754

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World Journal of Experimental Medicine

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2220-315x

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Exp Med. Jun 20, 2026; 16(2): 120754
Published online Jun 20, 2026. doi: 10.5493/wjem.v16.i2.120754

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Figure 1 Analogies between prolactin and peripartum cardiomyopathy, as well as the proposed axis of melatonin-prolactin-oxidative stress, which results in heart failure, emphasizing prolactin proteolysis. ROS: Reactive oxygen species; PRL: Prolactin; PPCM: Peripartum cardiomyopathy; HF: Heart failure. Original work, created on Microsoft PowerPoint, additionally using freely available elements from Servier Medical Art (https://smart.servier.com), licensed under CC BY 4.0 (https://creativecommons.org/Licenses/by/4.0/).

Citation: Savvidis C, Thomopoulos C, Ilias I. Melatonin supplementation, hyperprolactinemia, and incident heart failure: A proposed prolactin-mediated pathway for cardiovascular risk. World J Exp Med 2026; 16(2): 120754
URL: https://www.wjgnet.com/2220-315x/full/v16/i2/120754.htm
DOI: https://dx.doi.org/10.5493/wjem.v16.i2.120754

Savvidis C, Thomopoulos C, Ilias I. Melatonin supplementation, hyperprolactinemia, and incident heart failure: A proposed prolactin-mediated pathway for cardiovascular risk. World J Exp Med 2026; 16(2): 120754 [DOI: 10.5493/wjem.v16.i2.120754]

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