Aryl hydrocarbon receptor dynamics in esophageal squamous cell carcinoma: From immune modulation to therapeutic opportunities

doi:10.5493/wjem.v14.i3.96269

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Sep 20, 2024 (publication date) through Nov 29, 2024

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World Journal of Experimental Medicine

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2220-315x

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Exp Med. Sep 20, 2024; 14(3): 96269
Published online Sep 20, 2024. doi: 10.5493/wjem.v14.i3.96269

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Figure 1 Aryl hydrocarbon receptor signaling in esophageal squamous cell carcinoma development and immune escape. This figure illustrates the multifaceted influence of aryl hydrocarbon receptor (AhR) signaling on esophageal squamous cell carcinoma (ESCC) development and immune escape. Activation of AhR by environmental carcinogens or other ligands initiates a cascade of cellular processes that contribute to tumorigenesis. EMT: Epithelial-mesenchymal transition; NH3: Ammonia; PD-L1: Programmed death-ligand 1; ROS: Reactive oxygen species; TEPA: Triethylenetetramine.

Citation: Rahmati M, Moghtaderi H, Mohammadi S, Al-Harrasi A. Aryl hydrocarbon receptor dynamics in esophageal squamous cell carcinoma: From immune modulation to therapeutic opportunities. World J Exp Med 2024; 14(3): 96269
URL: https://www.wjgnet.com/2220-315x/full/v14/i3/96269.htm
DOI: https://dx.doi.org/10.5493/wjem.v14.i3.96269