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©2014 Baishideng Publishing Group Inc.
World J Exp Med. May 20, 2014; 4(2): 16-26
Published online May 20, 2014. doi: 10.5493/wjem.v4.i2.16
Published online May 20, 2014. doi: 10.5493/wjem.v4.i2.16
Table 1 Upregulation of extraembryonic phenotypes that could be involved in the different types of the wound healing reaction
| Phenotypes | Embryonicfunctions | Phases of theinflammatoryresponse | Phases of the wound healing reaction | |
| Extraembryonic phenotypes | Coelomic-amniotic axis | Nervous phase | Neurogenic systemic response | Stress response - Biogenic amines release |
| Sensitive and motor alterations | ||||
| Ischemia-reperfusion - Local oxidative and nitrosative stress | ||||
| Hydroelectrolytic alterations - Edema | ||||
| Inflammation blood cells - Coagulation | ||||
| Trophoblastic-vitelline axis | Immune phase | Bone-marrow related response | Enzymatic stress | |
| Corticosuprarenal hormones - Local storage | ||||
| Hematopoietic stem cells | ||||
| Mesenchymal stem cells | ||||
| Endothelial progenitor cells | ||||
| Embryonic phenotypes | Gastrulation | Angiogenic phase | Remodeling response | Myofibroblasts |
| Angiogenesis | ||||
| Endothelial egg | ||||
| Re-epithelization | ||||
| Fibrosis | ||||
- Citation: Aller MA, Arias JI, Arraez-Aybar LA, Gilsanz C, Arias J. Wound healing reaction: A switch from gestation to senescence. World J Exp Med 2014; 4(2): 16-26
- URL: https://www.wjgnet.com/2220-315X/full/v4/i2/16.htm
- DOI: https://dx.doi.org/10.5493/wjem.v4.i2.16