Klotho: A multifaceted protector in sepsis-induced organ damage and a potential therapeutic target

doi:10.5492/wjccm.v14.i3.103458

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Sep 9, 2025 (publication date) through Jul 18, 2025

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World Journal of Critical Care Medicine

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2220-3141

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Crit Care Med. Sep 9, 2025; 14(3): 103458
Published online Sep 9, 2025. doi: 10.5492/wjccm.v14.i3.103458

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Figure 1 The role of klotho administration after sepsis. FGF-R: Fibroblast growth factor-receptors; FoxO: Forkhead transcription factor O; HO-1: Heme oxygenase-1; ILs: Interleukins; MAPK: Mitogen activated protein kinase; Mn-SOD: Manganese-superoxide dismutase; NADPH: Nicotinamide adenine dinucleotide phosphate; NF-κB: nuclear factor-kappa β; Nrf-2: Nuclear factor erythroid related factor 2; PAR: Pathogen-associated receptor; ROS: Reactive oxygen species; TNF-α: Tumor necrosis factor-α; TWEAK: Tumor necrosis factor-related weak inducer of apoptosis.

Citation: Al-Kadi A, Anter A, Rofaeil RR, Sayed-Ahmed MM, Ahmed ASF. Klotho: A multifaceted protector in sepsis-induced organ damage and a potential therapeutic target. World J Crit Care Med 2025; 14(3): 103458
URL: https://www.wjgnet.com/2220-3141/full/v14/i3/103458.htm
DOI: https://dx.doi.org/10.5492/wjccm.v14.i3.103458