Copyright
©The Author(s) 2018.
World J Gastrointest Endosc. May 16, 2018; 10(5): 83-92
Published online May 16, 2018. doi: 10.4253/wjge.v10.i5.83
Published online May 16, 2018. doi: 10.4253/wjge.v10.i5.83
Figure 1 After endoscopic submucosal dissection endoscopic submucosal dissection, mean endoscopic submucosal dissection-induced ulcer areas at 4 and 8 wk in all patients (A), between Helicobacter pylori-positive patients and Helicobacter pylori-negative patients (B), between lansoprazole and vonoprazan (C), among patients with no atrophy, mild atrophy and severe atrophy (D), and among different locations of tumor (E).
ESD: endoscopic submucosal dissection; H. pylori: Helicobacter pylori.
Figure 2 After endoscopic submucosal dissection, mean reduction rate of endoscopic submucosal dissection-induced ulcer area at 4 wk in all patients (A), between Helicobacter pylori-positive and Helicobacter pylori-negative patients (B), between lansoprazole and vonoprazan (C), among CYP2C19 genotypes (EM, IM and PM) (D), among non-atrophy, mild atrophy and severe atrophy (E), and among different locations of tumor (lower third, middle third and upper third) (F).
H. pylori: Helicobacter pylori.
- Citation: Otsuka T, Sugimoto M, Ban H, Nakata T, Murata M, Nishida A, Inatomi O, Bamba S, Andoh A. Severity of gastric mucosal atrophy affects the healing speed of post-endoscopic submucosal dissection ulcers. World J Gastrointest Endosc 2018; 10(5): 83-92
- URL: https://www.wjgnet.com/1948-5190/full/v10/i5/83.htm
- DOI: https://dx.doi.org/10.4253/wjge.v10.i5.83